Nanoproteomic analysis of extracellular receptor kinase-1/2 post-translational activation in microdissected human hyperplastic colon lesions

Author:

Drew David A.12,Devers Thomas3,Horelik Nicole14,Yang Shi5,O'Brien Michael5,Wu Rong6,Rosenberg Daniel W.124

Affiliation:

1. Center for Molecular Medicine; University of Connecticut Health Center; Farmington Connecticut

2. Department of Genetics and Developmental Biology; University of Connecticut Health Center; Farmington CT USA

3. Division of Gastroenterology; University of Connecticut Health Center; Farmington CT USA

4. Colon Cancer Prevention Program; Neag Comprehensive Cancer Center; University of Connecticut Health Center; Farmington CT USA

5. Department of Pathology and Laboratory Medicine; Boston University School of Medicine; Boston MA USA

6. Connecticut Institute for Clinical and Translational Sciences; University of Connecticut Health Center; Farmington CT USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Molecular Biology,Biochemistry

Reference29 articles.

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2. Detection of aberrant crypt foci by magnifying colonoscopy;Yokota;Gastrointest. Endosc.,1997

3. Mutations in BRAF and KRAS differentially distinguish serrated versus non-serrated hyperplastic aberrant crypt foci in humans;Rosenberg;Cancer Res.,2007

4. Mutant KRAS in aberrant crypt foci (ACF): initiation of colorectal cancer;Pretlow;Biochimica et biophysica acta,2005

5. Investigation of the prevalence and number of aberrant crypt foci associated with human colorectal neoplasm;Sakai;Cancer Epidemiol. Biomarkers Prev.,2011

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