Glycine receptors are involved in hippocampal neuronal damage caused by oxygen-glucose deficiency
Author:
Affiliation:
1. Department of Cytology; O.O.Bogomoletz Institute of Physiology; National Academy of Sciences; Kiev Ukraine
2. Aix-Marseille University; INSERM, INS; Institut de Neurosciences des Systèmes; Marseille France
Funder
French Ministry of Foreign Affairs
Post-graduate Fellowship for Maleeva Galyna
Publisher
Wiley
Subject
Cell Biology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/cbin.11034/fullpdf
Reference51 articles.
1. Sustained NMDA receptor activation by spreading depolarizations can initiate excitotoxic injury in metabolically compromised neurons;Aiba;J Physiol,2012
2. Synaptic release and extracellular actions of Zn2+ limit propagation of spreading depression and related events in vitro and in vivo;Aiba;J Neurophysiol,2012
3. Oxygen and glucose deprivation induces mitochondrial dysfunction and oxidative stress in neurones but not in astrocytes in primary culture;Almeida;J Neurochem,2002
4. Age-related changes of glycine receptor at the rat hippocampus: from the embryo to the adult;Aroeira;J Neurochem,2011
5. Localization of glycine receptors in the human forebrain, brainstem, and cervical spinal cord: an immunohistochemical review;Baer;Front Mol Neurosci,2009
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