Discovery of an insulin‐induced gene binding compound that ameliorates nonalcoholic steatohepatitis by inhibiting sterol regulatory element‐binding protein–mediated lipogenesis-Reference-Cited by-同舟云学术

Discovery of an insulin‐induced gene binding compound that ameliorates nonalcoholic steatohepatitis by inhibiting sterol regulatory element‐binding protein–mediated lipogenesis

Published:2022-04-16 Issue:5 Volume:76 Page:1466-1481
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Jiang Shi‐You¹²,Yang Xinglin³,Yang Zimo³,Li Jue‐Wan²,Xu Meng‐Qiang²,Qu Yu‐Xiu²,Tang Jing‐Jie⁴,Li Yun‐Feng¹,Wang Liguo³,Shao Yi‐Wen⁵⁶,Meng Xin‐Yuan⁵⁶,Hu Huili⁵⁶,Song Bao‐Liang¹^ORCID,Rao Yu³,Qi Wei²

Affiliation:

1. Hubei Key Laboratory of Cell Homeostasis College of Life Sciences Wuhan University Wuhan China

2. Gene Editing Center School of Life Science and Technology ShanghaiTech University Shanghai China

3. MOE Key Laboratory of Protein Sciences School of Pharmaceutical Sciences MOE Key Laboratory of Bioorganic Phosphorus Chemistry & Chemical Biology Tsinghua University Beijing China

4. The State Key Laboratory of Molecular Biology Institute of Biochemistry and Cell Biology University of Chinese Academy of Sciences Chinese Academy of Sciences Shanghai China

5. The Key Laboratory of Experimental Teratology Ministry of Education and Department of Molecular Medicine and Genetics Shandong University Cheeloo Medical College School of Basic Medical Sciences Jinan China

6. The Research Center of Stem Cell and Regenerative Medicine Shandong University Cheeloo Medical College School of Basic Medical Sciences Jinan China

Funder

National Natural Science Foundation of China

Ministry of Science and Technology of the People's Republic of China

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.32381

Reference47 articles.

1. Human Fatty Liver Disease: Old Questions and New Insights

2. Increased Hepatic Synthesis and Dysregulation of Cholesterol Metabolism Is Associated with the Severity of Nonalcoholic Fatty Liver Disease