Targeting the arginine metabolic brake enhances immunotherapy for leukaemia-Reference-Cited by-同舟云学术

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Targeting the arginine metabolic brake enhances immunotherapy for leukaemia

Published:2019-01-11 Issue:8 Volume:145 Page:2201-2208
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

Mussai Francis¹^ORCID,Wheat Rachel¹,Sarrou Evgenia²,Booth Sarah¹,Stavrou Victoria¹,Fultang Livingstone¹,Perry Tracey³,Kearns Pamela³,Cheng Paul⁴,Keeshan Karen²,Craddock Charles³,De Santo Carmela¹

Affiliation:

1. Institute of Immunology and ImmunotherapyUniversity of Birmingham Birmingham United Kingdom

2. Paul O'Gorman Leukaemia Research Centre, College of Medicine, Veterinary Life SciencesInstitute of Cancer Sciences, University of Glasgow United Kingdom

3. Institute of Cancer and Genomic SciencesUniversity of Birmingham Birmingham United Kingdom

4. Bio‐cancer Treatment International Ltd Hong Kong

Funder

Bloodwise and Cancer Research UK

Treating Children with Cancer, University of Birmingham Alumni donors

Amber Phillpott Trust

Publisher

Wiley

Subject

Cancer Research,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ijc.32028

Reference25 articles.

1. International phase 3 study of azacitidine vs conventional care regimens in older patients with newly diagnosed AML with >30% blasts

2. Phase II Trial of Vorinostat With Idarubicin and Cytarabine for Patients With Newly Diagnosed Acute Myelogenous Leukemia or Myelodysplastic Syndrome

3. Cancer-testis antigen expression and its epigenetic modulation in acute myeloid leukemia

4. Acute myeloid leukemia creates an arginase-dependent immunosuppressive microenvironment

5. Outcome of Azacitidine Therapy in Acute Myeloid Leukemia Is not Improved by Concurrent Vorinostat Therapy but Is Predicted by a Diagnostic Molecular Signature

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