c-Jun blocks cell differentiation but not growth inhibition or apoptosis of chronic myelogenous leukemia cells induced by STI571 and by histone deacetylase inhibitors
Author:
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Reference41 articles.
1. The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells;Alani;Mol Cell Biol,1991
2. The chronic myelogenous leukemia-specific p210 protein is the product of the bcr-abl hybrid gene;Ben-Neriah;Science,1986
3. Detection of BCR-ABL mutations in patients with CML treated with imatinib is virtually always accompanied by clinical resistance, and mutations in the ATP phosphate-binding loop (P-loop) are associated with a poor prognosis;Branford;Blood,2003
4. Suppression of oncogene-induced transformation by a deletion mutant of c-jun;Brown;Oncogene,1992
5. Inhibition of the Abl protein-tyrosine kinase in vitro and in vivo by a 2-phenylaminopyrimidine derivative;Buchdunger;Cancer Res,1996
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