Differential Kv1.3, KCa3.1, and Kir2.1 expression in “classically” and “alternatively” activated microglia-Reference-Cited by-同舟云学术

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Differential Kv1.3, KCa3.1, and Kir2.1 expression in “classically” and “alternatively” activated microglia

Published:2016-10-03 Issue:1 Volume:65 Page:106-121
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Nguyen Hai M.¹,Grössinger Eva M.¹,Horiuchi Makoto²³,Davis Kyle W.²,Jin Lee-Way²³,Maezawa Izumi²³,Wulff Heike¹

Affiliation:

1. Department of Pharmacology; University of California; Davis California

2. Department of Pathology and Laboratory Medicine; University of California Davis Medical Center; Sacramento California

3. M.I.N.D. Institute, University of California Davis Medical Center; Davis, Sacramento California

Funder

National Institute of Neurological Disorders and Stroke

National Institute on Aging Award

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/glia.23078/fullpdf

Reference69 articles.

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2. Targeting effector memory T cells with the small molecule Kv1.3 blocker PAP-1 suppresses allergic contact dermatitis;Azam;J Invest Dermatol,2007

3. Lipopolysaccharide-induced down-regulation of Ca2+ release-activated Ca2+ currents (I CRAC) but not Ca2+-activated TRPM4-like currents (I CAN) in cultured mouse microglial cells;Beck;J Physiol Lond,2008

4. Potassium channels, memory T cells and multiple sclerosis;Beeton;Neuroscientist

5. Selective blockade of T lymphocyte K+ channels ameliorates experimental autoimmune encephalomyelitis, a model for multiple sclerosis;Beeton;Proc Natl Acad Sci USA,2001

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