T lymphocyte development in p56lck deficient mice: allelic exclusion of the TcR β locus is incomplete but thymocyte development is not restored by TcR β or TcR αβ transgenes-Reference-Cited by-同舟云学术

T lymphocyte development in p56lck deficient mice: allelic exclusion of the TcR β locus is incomplete but thymocyte development is not restored by TcR β or TcR αβ transgenes

Published:1995-05 Issue:5 Volume:25 Page:1312-1318
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Wallace Valerie A.,Kawai Kazuhiro,Levelt Christiaan N.,Kishihara Kenji,Molina Thierry,Timms Emma,Pircher Hanspeter,Penninger Josef,Ohashi Pamela S.,Eichmann Klaus,Mak Tak W.

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference42 articles.

1. The CD4 and CD8 T cell surface antigens are associated with the internal membrane tyrosine-protein kinase p56lck

2. The CD4 receptor is complexed in detergent lysates to a protein-tyrosine kinase (pp58) from human T lymphocytes.

3. Signal transduction by lymphocyte antigen receptors

4. Inability of CD8α′polypeptides to associate with p56lck correlates with impaired function in vitro and lack of expression in vivo

5. A signaling role for the cytoplasmic segment of the CD8 alpha chain detected under limiting stimulatory conditions.

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