Identification of Cadherin 11 as a Mediator of Dermal Fibrosis and Possible Role in Systemic Sclerosis-Reference-Cited by-同舟云学术

Identification of Cadherin 11 as a Mediator of Dermal Fibrosis and Possible Role in Systemic Sclerosis

Published:2014-03-28 Issue:4 Volume:66 Page:1010-1021
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Wu Minghua¹,Pedroza Mesias²,Lafyatis Robert³,George Anuh-Teresa²,Mayes Maureen D.¹,Assassi Shervin¹,Tan Filemon K.¹,Brenner Michael B.⁴,Agarwal Sandeep K.⁵

Affiliation:

1. University of Texas Health Science Center at; Houston

2. Baylor College of Medicine; Houston Texas

3. Boston University School of Medicine; Boston Massachusetts

4. Brigham and Women's Hospital and Harvard Medical School; Boston Massachusetts

5. University of Texas Health Science Center at Houston and Baylor College of Medicine; Houston Texas

Funder

NIH (National Institute of Arthritis and Musculoskeletal and Skin Diseases [NIAMS])

Scleroderma Foundation New Investigator Award

NIAMS

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Reference48 articles.

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3. Targeted disruption of TGF-β/Smad3 signaling modulates skin fibrosis in a mouse model of scleroderma;Lakos;Am J Pathol,2004

4. Signatures of differentially regulated interferon gene expression and vasculotrophism in the peripheral blood cells of systemic sclerosis patients;Tan;Rheumatology (Oxford),2006

5. Toll-like receptor 3 upregulation by type I interferon in healthy and scleroderma dermal fibroblasts;Agarwal;Arthritis Res Ther,2011

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