Targeted Disruption of TGF-β/Smad3 Signaling Modulates Skin Fibrosis in a Mouse Model of Scleroderma-Reference-Cited by-同舟云学术

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Targeted Disruption of TGF-β/Smad3 Signaling Modulates Skin Fibrosis in a Mouse Model of Scleroderma

Published:2004-07 Issue:1 Volume:165 Page:203-217
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Lakos Gabriella,Takagawa Shinsuke,Chen Shu-Jen,Ferreira Ahalia M.,Han Gangwen,Masuda Koichi,Wang Xiao-Jing,DiPietro Luisa A.,Varga John

Funder

Uehara Memorial Foundation

Arthritis Foundation

Scleroderma Foundation

National Institutes of Health

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference64 articles.

1. Fibrosis in scleroderma;Kissin;Rheum Dis Clin North Am,2003

2. Myofibroblasts from scleroderma skin synthesize elevated levels of collagen and tissue inhibitor of metalloproteinase (TIMP-1) with two forms of TIMP-1;Kirk;J Biol Chem,1995

3. Molecular and cell biology of TGF-beta;Roberts;Miner Electrolyte Metab,1998

4. Role of transforming growth factor β in human disease;Blobe;N Engl J Med,2000

5. SMADs: mediators and regulators of TGF-β signaling;Kretzschmar;Curr Opin Genet Dev,1998

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