Carbohydrate response element‐binding protein regulates lipid metabolism via mTOR complex1 in diabetic nephropathy
Author:
Affiliation:
1. Department of Pathology Hebei Medical University Shijiazhuang China
2. Hebei Key Laboratory of Kidney Disease Shijiazhuang China
3. Department of Nephrology, Second Hospital Hebei Medical University Shijiazhuang China
Funder
Natural Science Foundation of Hebei Province
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.29890
Reference40 articles.
1. ChREBP Mediates Glucose Repression of Peroxisome Proliferator-activated Receptor α Expression in Pancreatic β-Cells
2. WBSCR14, a gene mapping to the Williams-Beuren syndrome deleted region, is a new member of the Mlx transcription factor network
3. Disulfide-bond A oxidoreductase-like protein protects against ectopic fat deposition and lipid-related kidney damage in diabetic nephropathy
4. Carbohydrate response element binding protein (ChREBP) modulates the inflammatory response of mesangial cells in response to glucose
5. Interaction of PPARα With the Canonic Wnt Pathway in the Regulation of Renal Fibrosis
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