Expression of paramyxovirus V proteins promotes replication and spread of hepatitis C virus in cultures of primary human fetal liver cells-Reference-Cited by-全球学者库

Expression of paramyxovirus V proteins promotes replication and spread of hepatitis C virus in cultures of primary human fetal liver cells

Published:2011-12 Issue:6 Volume:54 Page:1901-1912
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Andrus Linda¹,Marukian Svetlana¹,Jones Christopher T.¹,Catanese Maria Teresa¹,Sheahan Timothy P.¹,Schoggins John W.¹,Barry Walter T.¹,Dustin Lynn B.¹,Trehan Kartik²,Ploss Alexander¹,Bhatia Sangeeta N.²,Rice Charles M.¹

Affiliation:

1. Center for the Study of Hepatitis C, Laboratory of Virology and Infectious Diseases, The Rockefeller University, New York, NY

2. Division of Health Sciences and Technology, Department of Electrical Engineering and Computer Science, Howard Hughes Medical Institute, Massachusetts Institute of Technology, Cambridge, MA

Publisher

Wiley

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.24557

Reference40 articles.

1. Recovery, Persistence, and Sequelae in Hepatitis C Virus Infection: A Perspective on Long-Term Outcome

2. Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity

3. Interaction between the HCV NS3 protein and the host TBK1 protein leads to inhibition of cellular antiviral responses

4. Hepatitis C Virus Core Protein Blocks Interferon Signaling by Interaction with the STAT1 SH2 Domain

5. Evasion of intracellular host defence by hepatitis C virus

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