<i> <scp>FGFR2</scp> :: <scp>TACC2</scp> </i> fusion as a novel <scp>KIT</scp> ‐independent mechanism of targeted therapy failure in a multidrug‐resistant gastrointestinal stromal tumor-Reference-Cited by-同舟云学术

FGFR2 :: TACC2 fusion as a novel KIT ‐independent mechanism of targeted therapy failure in a multidrug‐resistant gastrointestinal stromal tumor

Published:2022-02-22 Issue:7 Volume:61 Page:412-419
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosomes & Cancer

Author:

Dermawan Josephine K.¹^ORCID,Vanderbilt Chad M.¹,Chang Jason C.¹,Untch Brian R.²,Singer Samuel²,Chi Ping³,Tap William D.³,Antonescu Cristina R.¹^ORCID

Affiliation:

1. Department of Pathology Memorial Sloan Kettering Cancer Center New York New York USA

2. Department of Surgery Memorial Sloan Kettering Cancer Center New York New York USA

3. Department of Medicine Memorial Sloan Kettering Cancer Center New York New York USA

Publisher

Wiley

Subject

Cancer Research,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/gcc.23030

Reference54 articles.

1. Natural History of Imatinib-naive GISTs

2. Gain-of-Function Mutations of c- kit in Human Gastrointestinal Stromal Tumors

3. PDGFRA Activating Mutations in Gastrointestinal Stromal Tumors

4. SDHA mutations in adult and pediatric wild-type gastrointestinal stromal tumors

5. Recurrent epimutation of SDHC in gastrointestinal stromal tumors

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