Recurrent epimutation of SDHC in gastrointestinal stromal tumors

Author:

Killian J. Keith1,Miettinen Markku2,Walker Robert L.1,Wang Yonghong1,Zhu Yuelin Jack1,Waterfall Joshua J.1,Noyes Natalia1,Retnakumar Parvathy1,Yang Zhiming2,Smith William I.3,Killian M. Scott4,Lau C. Christopher1,Pineda Marbin1,Walling Jennifer1,Stevenson Holly1,Smith Carly5,Wang Zengfeng2,Lasota Jerzy2,Kim Su Young6,Boikos Sosipatros A.6,Helman Lee J.6,Meltzer Paul S.1

Affiliation:

1. Genetics Branch, National Cancer Institute, National Institutes of Health (NIH), Bethesda, MD 20892, USA.

2. Laboratory of Pathology, Center for Cancer Research, NIH, Bethesda, MD 20892, USA.

3. Department of Pathology, Suburban Hospital, Bethesda, MD 20814, USA.

4. University of South Dakota, Vermillion, SD 57069, USA.

5. National Heart Lung and Blood Institute, NIH, Bethesda, MD 20892, USA.

6. Pediatric Oncology Branch, Center for Cancer Research, NIH, Bethesda, MD 20892, USA.

Abstract

Methylation of the SDH gene explains the loss of SDH gene expression in SDH wild-type gastrointestinal stromal tumors.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

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