Expression of the glucose-sensing receptor T1R3 in pancreatic islet: changes in the expression levels in various nutritional and metabolic states-Reference-Cited by-同舟云学术

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Expression of the glucose-sensing receptor T1R3 in pancreatic islet: changes in the expression levels in various nutritional and metabolic states

Published:2014 Issue:8 Volume:61 Page:797-805
ISSN:0918-8959
Container-title:Endocrine Journal
language:en
Short-container-title:Endocr J

Author:

Medina Anya¹,Nakagawa Yuko¹,Ma Jinhui¹,Li Longfei¹,Hamano Kunihisa¹,Akimoto Toshio²,Ninomiya Yuzo³,Kojima Itaru¹

Affiliation:

1. Institute for Molecular & Cellular Regulation, Gunma University, Maebashi 371-8512, Japan

2. Division of Laboratory Animal Science, Nippon Medical School, Tokyo 113-8602, Japan

3. Department of Oral Physiology, Kyushu University Graduate School of Dental Science, Fukuoka 812-8582, Japan

Publisher

Japan Endocrine Society

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Link

https://www.jstage.jst.go.jp/article/endocrj/61/8/61_EJ14-0221/_pdf

Reference25 articles.

1. 1. Prentki M, Matschinsky FM (1987) Ca²⁺, cAMP and phospholipid derived messengers in coupling mechanism of insulin secretion. Physiol Rev 67: 1185-1248.

2. 2. Matschinsky FM (1990) Glucokinase as glucose sensor and metabolic signal generation in pancreatic β-cells and hepatocytes. Diabetes 39: 647-652.

3. 3. Eto K, Tsubamoto Y, Terauchi Y, Sugiyama T, Kishimoto T, et al. (1999) Role of NADH shuttle system in Glucose-induced activation of mitochondrial metabolism and insulin secretion. Science 283: 981-985.

4. 4. Farfari S, Schulz V, Corkey B, Prentki M (2000) Glucose-regulated anaplerosis and cataplerosis in pancreatic β-cells. Diabetes 49: 718-726.

5. 5. Temussi PA (2009) Sweet, bitter and umami receptors: a complex relationship. Trend Biochem Sci 34: 296-302.

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