Evidence that Unrejoined DNA Double-Strand Breaks are not Predominantly Responsible for Chromosomal Radiosensitivity of AT Fibroblasts
Author:
Publisher
Radiation Research Society
Subject
Radiology, Nuclear Medicine and imaging,Radiation,Biophysics
Cited by 14 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Live-cell tracking of γ-H2AX kinetics reveals the distinct modes of ATM and DNA-PK in the immediate response to DNA damage;Journal of Cell Science;2023-04-15
2. Live-Cell Tracking of γ-H2AX Kinetics Reveals the Distinct Modes of ATM and DNA-PK in Immediate Response to DNA Damage;2022-10-09
3. Straightening Beta: Overdispersion of Lethal Chromosome Aberrations following Radiotherapeutic Doses Leads to Terminal Linearity in the Alpha–Beta Model;Frontiers in Oncology;2017-12-21
4. How cancer cells hijack DNA double-strand break repair pathways to gain genomic instability;Biochemical Journal;2015-09-21
5. From DNA damage to chromosome aberrations: Joining the break;Mutation Research/Genetic Toxicology and Environmental Mutagenesis;2013-08
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