Menin‐regulated Pbk controls high fat diet‐induced compensatory beta cell proliferation-Reference-Cited by-同舟云学术

Menin‐regulated Pbk controls high fat diet‐induced compensatory beta cell proliferation

Published:2021-04-06 Issue:5 Volume:13 Page:
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Ma Jian¹²^ORCID,Xing Bowen¹,Cao Yan¹²,He Xin¹^ORCID,Bennett Kate E³,Tong Chao⁴,An Chiying¹²,Hojnacki Taylor¹,Feng Zijie¹,Deng Sunbin⁵,Ling Sunbin¹,Xie Gengchen¹,Wu Yuan¹,Ren Yue⁶,Yu Ming²,Katona Bryson W¹³,Li Hongzhe⁶,Naji Ali²,Hua Xianxin¹²^ORCID

Affiliation:

1. Department of Cancer Biology Abramson Family Cancer Research Institute University of Pennsylvania Perelman School of Medicine Philadelphia PA USA

2. Institute for Diabetes, Obesity, and Metabolism University of Pennsylvania Perelman School of Medicine Philadelphia PA USA

3. Division of Gastroenterology University of Pennsylvania Perelman School of Medicine Philadelphia PA USA

4. Department of Biology University of Pennsylvania Philadelphia PA USA

5. Department of Chemistry University of Pennsylvania Philadelphia PA USA

6. Department of Biochemistry and Biophysics University of Pennsylvania Perelman School of Medicine Philadelphia PA USA

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.202013524

Reference68 articles.

1. Cloning and Expression of a Novel MAPKK-like Protein Kinase, Lymphokine-activated Killer T-cell-originated Protein Kinase, Specifically Expressed in the Testis and Activated Lymphoid Cells

2. A Mitotic Kinase TOPK Enhances Cdk1/cyclin B1-dependent Phosphorylation of PRC1 and Promotes Cytokinesis

3. Menin Interacts with the AP1 Transcription Factor JunD and Represses JunD-Activated Transcription

4. Treatment of arthritis with a selective inhibitor of c-Fos/activator protein-1

5. PBK/TOPK promotes tumour cell proliferation through p38 MAPK activity and regulation of the DNA damage response

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