Mammalian UPF3A and UPF3B can activate nonsense‐mediated mRNA decay independently of their exon junction complex binding-Reference-Cited by-同舟云学术

Mammalian UPF3A and UPF3B can activate nonsense‐mediated mRNA decay independently of their exon junction complex binding

Published:2022-04-22 Issue:10 Volume:41 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:The EMBO Journal

Author:

Yi Zhongxia¹²^ORCID,Arvola René M¹²^ORCID,Myers Sean³,Dilsavor Corinne N²^ORCID,Abu Alhasan Rabab¹²^ORCID,Carter Bayley N²^ORCID,Patton Robert D¹³,Bundschuh Ralf¹³⁴⁵,Singh Guramrit¹²^ORCID

Affiliation:

1. Center for RNA Biology The Ohio State University Columbus OH USA

2. Department of Molecular Genetics The Ohio State University Columbus OH USA

3. Department of Physics The Ohio State University Columbus OH USA

4. Department of Chemistry and Biochemistry The Ohio State University Columbus OH USA

5. Division of Hematology Department of Internal Medicine The Ohio State University Columbus OH USA

Funder

National Institutes of Health

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2021109202

Reference76 articles.

1. A novel frameshift mutation in UPF3B identified in brothers affected with childhood onset schizophrenia and autism spectrum disorders

2. Full UPF3B function is critical for neuronal differentiation of neural stem cells

3. A faux 3′-UTR promotes aberrant termination and triggers nonsense- mediated mRNA decay

4. Drosophila Upf1 and Upf2 loss of function inhibits cell growth and causes animal death in a Upf3-independent manner

5. ICE1 promotes the link between splicing and nonsense-mediated mRNA decay

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