NF‐κB activation in astrocytes drives a stage‐specific beneficial neuroimmunological response in ALS-Reference-Cited by-同舟云学术

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NF‐κB activation in astrocytes drives a stage‐specific beneficial neuroimmunological response in ALS

Published:2018-06-06 Issue:16 Volume:37 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Ouali Alami Najwa¹,Schurr Christine²,Olde Heuvel Florian¹,Tang Linyun¹,Li Qian¹^ORCID,Tasdogan Alpaslan³,Kimbara Atsushi⁴,Nettekoven Matthias⁴,Ottaviani Giorgio⁴,Raposo Catarina⁴,Röver Stephan⁴,Rogers‐Evans Mark⁴,Rothenhäusler Benno⁴,Ullmer Christoph⁴,Fingerle Jürgen⁵,Grether Uwe⁴,Knuesel Irene⁴,Boeckers Tobias M⁶,Ludolph Albert¹,Wirth Thomas²,Roselli Francesco¹⁶^ORCID,Baumann Bernd²

Affiliation:

1. Department of Neurology Ulm University Ulm Germany

2. Institute of Physiological Chemistry Ulm University Ulm Germany

3. Institute of Immunology Ulm University Ulm Germany

4. Roche Pharma Research and Early Development Roche Innovation Center Basel F. Hoffmann‐La Roche Ltd. Basel Switzerland

5. Natural and Medical Sciences Institute Tübingen University Reutlingen Germany

6. Department of Anatomy and Cell Biology Ulm University Ulm Germany

Funder

Universität Ulm

F. Hoffmann-La Roche

Deutsche Forschungsgemeinschaft

Ministerium für Wissenschaft, Forschung und Kunst Baden-Württemberg

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201798697

Reference63 articles.

1. Constitutive IKK2 activation in acinar cells is sufficient to induce pancreatitis in vivo

2. CD4+ T cells support glial neuroprotection, slow disease progression, and modify glial morphology in an animal model of inherited ALS

3. Endogenous regulatory T lymphocytes ameliorate amyotrophic lateral sclerosis in mice and correlate with disease progression in patients with amyotrophic lateral sclerosis

4. TOM40 Mediates Mitochondrial Dysfunction Induced by α-Synuclein Accumulation in Parkinson’s Disease

5. New tools for studying microglia in the mouse and human CNS

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