Too much death can kill you: inhibiting intrinsic apoptosis to treat disease-Reference-Cited by-同舟云学术

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Too much death can kill you: inhibiting intrinsic apoptosis to treat disease

Published:2021-05-26 Issue:14 Volume:40 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Li Kaiming¹²^ORCID,Delft Mark F¹²^ORCID,Dewson Grant¹²^ORCID

Affiliation:

1. The Walter and Eliza Hall Institute of Medical Research Melbourne VIC Australia

2. Department of Medical Biology University of Melbourne Royal Parade Melbourne VIC Australia

Funder

National Health and Medical Research Council

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2020107341

Reference228 articles.

1. The Bcl-2 apoptotic switch in cancer development and therapy

2. OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28

3. Cell death of chondrocytes is a combination between apoptosis and autophagy during the pathogenesis of Osteoarthritis within an experimental model

4. A small-molecule allosteric inhibitor of BAX protects against doxorubicin-induced cardiomyopathy

5. Cytomegalovirus cell death suppressor vMIA blocks Bax- but not Bak-mediated apoptosis by binding and sequestering Bax at mitochondria

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3. RVFV virulence factor NSs triggers the mitochondrial MCL-1-BAK axis to activate pathogenic NLRP3 pyroptosis;PLOS Pathogens;2024-08-30

4. Study on the sensitizing effect of SM-1 combined with irradiation on head and neck squamous cell carcinoma;International Journal of Radiation Biology;2024-08-13

5. Differential regulation of BAX and BAK apoptotic activity revealed by a novel small molecule;2024-08-05

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