CRISPR screens are feasible in TP 53 wild‐type cells-Reference-Cited by-同舟云学术

CRISPR screens are feasible in TP 53 wild‐type cells

Published:2019-08 Issue:8 Volume:15 Page:
ISSN:1744-4292
Container-title:Molecular Systems Biology
language:en
Short-container-title:Mol Syst Biol

Author:

Brown Kevin R¹^ORCID,Mair Barbara¹^ORCID,Soste Martin¹^ORCID,Moffat Jason¹²³^ORCID

Affiliation:

1. Donnelly Centre University of Toronto Toronto ON Canada

2. Department of Molecular Genetics University of Toronto Toronto ON Canada

3. Canadian Institute for Advanced Research Toronto ON Canada

Publisher

EMBO

Subject

Applied Mathematics,Computational Theory and Mathematics,General Agricultural and Biological Sciences,General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Information Systems

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/msb.20188679

Reference17 articles.

1. Epigenetic and genetic features of 24 colon cancer cell lines

2. DNA damage triggers a prolonged p53-dependent G1 arrest and long-term induction of Cip1 in normal human fibroblasts.

3. Optimized sgRNA design to maximize activity and minimize off-target effects of CRISPR-Cas9

4. Mutational processes shape the landscape of TP53 mutations in human cancer

5. CRISPR–Cas9 genome editing induces a p53-mediated DNA damage response

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