Pathway connectivity and signaling coordination in the yeast stress‐activated signaling network-Reference-Cited by-同舟云学术

Pathway connectivity and signaling coordination in the yeast stress‐activated signaling network

Published:2014-11 Issue:11 Volume:10 Page:759
ISSN:1744-4292
Container-title:Molecular Systems Biology
language:en
Short-container-title:Mol Syst Biol

Author:

Chasman Deborah¹,Ho Yi‐Hsuan²,Berry David B²,Nemec Corey M³,MacGilvray Matthew E²,Hose James²,Merrill Anna E⁴,Lee M Violet⁴,Will Jessica L²,Coon Joshua J⁴⁵⁶,Ansari Aseem Z³⁵,Craven Mark¹⁵⁷,Gasch Audrey P²⁵

Affiliation:

1. Department of Computer Sciences University of Wisconsin‐Madison Madison WI USA

2. Laboratory of Genetics University of Wisconsin‐Madison Madison WI USA

3. Department of Biochemistry University of Wisconsin‐Madison Madison WI USA

4. Department of Chemistry University of Wisconsin‐Madison Madison WI USA

5. Genome Center of Wisconsin University of Wisconsin‐Madison Madison WI USA

6. Department of Biological Chemistry University of Wisconsin‐Madison Madison WI USA

7. Department of Biostatistics and Medical Informatics University of Wisconsin‐Madison Madison WI USA

Publisher

EMBO

Subject

Applied Mathematics,Computational Theory and Mathematics,General Agricultural and Biological Sciences,General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Information Systems

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/msb.20145120

Reference138 articles.

1. YEASTRACT: providing a programmatic access to curated transcriptional regulatory associations in Saccharomyces cerevisiae through a web services interface

2. Time-Dependent Quantitative Multicomponent Control of the G 1 -S Network by the Stress-Activated Protein Kinase Hog1 upon Osmostress

3. The histone deacetylase Rpd3p is required for transient changes in genomic expression in response to stress

4. Osmostress-induced transcription by Hot1 depends on a Hog1-mediated recruitment of the RNA Pol II

5. Regulation of Cell Cycle Progression by Swe1p and Hog1p Following Hypertonic Stress

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