The molecular architecture of cell cycle arrest-Reference-Cited by-同舟云学术

The molecular architecture of cell cycle arrest

Published:2022-09 Issue:9 Volume:18 Page:
ISSN:1744-4292
Container-title:Molecular Systems Biology
language:en
Short-container-title:Molecular Systems Biology

Author:

Stallaert Wayne¹²^ORCID,Taylor Sovanny R¹²,Kedziora Katarzyna M¹³,Taylor Colin D¹²^ORCID,Sobon Holly K¹²,Young Catherine L¹²,Limas Juanita C⁴,Varblow Holloway Jonah¹²,Johnson Martha S⁴^ORCID,Cook Jeanette Gowen⁴⁵^ORCID,Purvis Jeremy E¹²^ORCID

Affiliation:

1. Department of Genetics University of North Carolina at Chapel Hill Chapel Hill NC USA

2. Computational Medicine Program University of North Carolina at Chapel Hill Chapel Hill NC USA

3. Bioinformatics and Analytics Research Collaborative (BARC) University of North Carolina at Chapel Hill Chapel Hill NC USA

4. Department of Biochemistry and Biophysics University of North Carolina at Chapel Hill Chapel Hill NC USA

5. Department of Pharmacology University of North Carolina at Chapel Hill Chapel Hill NC USA

Funder

Chan Zuckerberg Initiative

National Institutes of Health

Howard Hughes Medical Institute

National Science Foundation

Publisher

EMBO

Subject

Applied Mathematics,Computational Theory and Mathematics,General Agricultural and Biological Sciences,General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Information Systems

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/msb.202211087

Reference62 articles.

1. Endogenous Replication Stress in Mother Cells Leads to Quiescence of Daughter Cells

2. DNA damage during S-phase mediates the proliferation-quiescence decision in the subsequent G1 via p21 expression

3. Retinoblastoma protein and anaphase-promoting complex physically interact and functionally cooperate during cell-cycle exit

4. EVIDENCE FOR AN ESSENTIALLY CONSTANT DURATION OF DNA SYNTHESIS IN RENEWING EPITHELIA OF THE ADULT MOUSE

5. Attractor Landscape Analysis Reveals Feedback Loops in the p53 Network That Control the Cellular Response to DNA Damage

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