Aminoguanidine Inhibits the AGE–RAGE Axis to Modulate the Induction of Periodontitis but Has Limited Effects on the Progression and Recovery of Experimental Periodontitis: A Preliminary Study
Author:
Publisher
Wiley
Subject
Periodontics
Link
http://www.joponline.org/doi/pdf/10.1902/jop.2013.130238
Reference33 articles.
1. The relationship between receptor for advanced glycation end products expression and the severity of periodontal disease in the gingiva of diabetic and non diabetic periodontitis patients
2. Receptor for Advanced Glycation End Products, Inflammation, and Accelerated Periodontal Disease in Diabetes: Mechanisms and Insights Into Therapeutic Modalities
3. Diabetes Mellitus and Periodontal Diseases
4. Blockade of RAGE suppresses periodontitis-associated bone loss in diabetic mice
5. Progression of Periodontal Destruction and the Roles of Advanced Glycation End Products in Experimental Diabetes
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3. Allantoin, dipotassium glycyrrhizinate, and azulene sulfonate sodium hydrate inhibit gingival inflammation induced by advanced glycation end products via antiglycation effects <i>in vitro</i>;Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology);2022-03-31
4. Pathogenic Molecular Mechanisms in Periodontitis and Peri-Implantitis: Role of Advanced Glycation End Products;Life;2022-01-30
5. Modulation of periodontitis progression using pH ‐responsive nanosphere encapsulating metronidazole or N ‐phenacylthialzolium bromide;Journal of Periodontal Research;2017-08-10
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