Inhibition by KB-R7943 of the Reverse Mode of the Na+/Ca2+ Exchanger Reduces Ca2+ Overload in Ischemic-Reperfused Rat Hearts.
Author:
Affiliation:
1. Division of Cardiology, Department of Internal Medicine, Aoto Hospital, The Jikei University, School of Medicine
Publisher
Japanese Circulation Society
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
http://www.jstage.jst.go.jp/article/circj/66/4/66_4_390/_pdf
Reference41 articles.
1. Generation of Free Radicals and the Damage Done to the Sarcoplasmic Reticulum During Reperfusion Injury Following Brief Ischemia in the Canine Heart
2. Postischemic Reperfusion Injury Can Be Attenuated by Oxygen Tension Control
3. Role of Cardiac ATP-Sensitive K+ Channels Induced by Angiotensin II Type 1 Receptor Antagonist on Metabolism, Contraction and Relaxation in Ischemia-Reperfused Rabbit Heart.
4. Role of intracellular Na+ in Ca2+ overload and depressed recovery of ventricular function of reperfused ischemic rat hearts. Possible involvement of H+-Na+ and Na+-Ca2+ exchange.
5. Elevation in cytosolic free calcium concentration early in myocardial ischemia in perfused rat heart.
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