Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI-Reference-Cited by-同舟云学术

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Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

Published:2016-03-21 Issue:11 Volume:27 Page:3356-3367
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Lan Rongpei,Geng Hui,Singha Prajjal K.,Saikumar Pothana,Bottinger Erwin P.,Weinberg Joel M.,Venkatachalam Manjeri A.

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference65 articles.

1. Recent Advances in the Pathophysiology of Ischemic Acute Renal Failure

2. Inhibition of Autoregulated TGFβ Signaling Simultaneously Enhances Proliferation and Differentiation of Kidney Epithelium and Promotes Repair Following Renal Ischemia

3. Lysophosphatidic Acid Increases Proximal Tubule Cell Secretion of Profibrotic Cytokines PDGF-B and CTGF through LPA2- and Gαq-Mediated Rho and αvβ6 Integrin-Dependent Activation of TGF-β

4. PTEN loss defines a TGF-β-induced tubule phenotype of failed differentiation and JNK signaling during renal fibrosis

5. Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury

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