Novel signaling pathways that regulate suppression of insulin gene expression in type 2 diabetes
Author:
Affiliation:
1. Department of Life Sciences, Faculty of Agriculture, Kagawa University
2. Laboratory of Molecular Biology, Science Research Center, Kochi Medical School
3. Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University
Publisher
Japanese Electrophoresis Society
Link
https://www.jstage.jst.go.jp/article/electroph/65/2/65_47/_pdf
Reference9 articles.
1. 1) Park KG, Lee KM, Seo HY, et al. Glucotoxicity in the INS-1 rat insulinoma cell line is mediated by the orphan nuclear receptor small heterodimer partner. Diabetes. 2007;56:431–437.
2. 2) Ubeda M, Rukstalis JM, Habener JF. Inhibition of cyclin-dependent kinase 5 activity protects pancreatic beta cells from glucotoxicity. J Biol Chem. 2006;281:28858–28864.
3. 3) 杉山康憲，亀下 勇．第67回総会シンポジウム：電気泳動を基盤とした研究の現状と今後の展望．電気泳動．2017;61:1–5.
4. 4) Sugiyama Y, Murao K, Imachi H, et al. Calcium/calmodulin-dependent protein kinase IV involvement in the pathophysiology of glucotoxicity in rat pancreatic β-cells. Metabolism. 2011;60:145–153.
5. 5) Ban N, Yamada Y, Someya Y, et al. Activating transcription factor-2 is a positive regulator in CaM kinase IV-induced human insulin gene expression. Diabetes. 2000;49:1142–1148.
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