Calcium/calmodulin-dependent protein kinase IV involvement in the pathophysiology of glucotoxicity in rat pancreatic β-cells-Reference-Cited by-同舟云学术

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Calcium/calmodulin-dependent protein kinase IV involvement in the pathophysiology of glucotoxicity in rat pancreatic β-cells

Published:2011-01 Issue:1 Volume:60 Page:145-153
ISSN:0026-0495
Container-title:Metabolism
language:en
Short-container-title:Metabolism

Author:

Sugiyama Yasunori,Murao Koji,Imachi Hitomi,Sueyoshi Noriyuki,Ishida Toshihiko,Kameshita Isamu

Funder

Scientific Research from the Ministry of Education, Culture, Sports, Science, and Technology of Japan;

Research Fellowships of the Japan Society for the Promotion of Science for Young Scientists;

Fund for Kagawa University Young Scientists

Publisher

Elsevier BV

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Reference45 articles.

1. Control of insulin gene expression in pancreatic beta-cells and in an insulin-producing cell line, RIN-5F cells. II. Regulation of insulin mRNA stability;Welsh;J Biol Chem,1985

2. Protective mechanism of glucose against alloxan-induced pancreatic beta-cell damage;Park;Biochem Biophys Res Commun,1995

3. Reduction of insulin gene transcription in HIT-T15 beta cells chronically exposed to a supraphysiologic glucose concentration is associated with loss of STF-1 transcription factor expression;Olson;Proc Natl Acad Sci USA,1995

4. Chronic hyperglycaemia is associated with impaired glucose influence on insulin secretion. A study in normal rats using chronic in vivo glucose infusions;Leahy;J Clin Investig,1986

5. Glucotoxicity and beta-cell failure in type 2 diabetes mellitus;Kaiser;J Pediatr Endocrinol Metab,2003

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