Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension-Reference-Cited by-同舟云学术

Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension

Published:2019-07-15 Issue:16 Volume:597 Page:4325-4340
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Ma Huijie¹²,Chen Shao‐Rui¹,Chen Hong¹,Pan Hui‐Lin¹^ORCID

Affiliation:

1. Center for Neuroscience and Pain ResearchDepartment of Anesthesiology and Perioperative MedicineUniversity of Texas MD Anderson Cancer Center Houston TX 77030 USA

2. Department of PhysiologyHebei Medical University Shijiazhuang Hebei 050017 China

Funder

National Science Foundation

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP278427

Reference63 articles.

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2. Inhibition of the Hypothalamic Paraventricular Nucleus in Spontaneously Hypertensive Rats Dramatically Reduces Sympathetic Vasomotor Tone

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4. Hemodynamic Response to Gabapentin in Conscious Spontaneously Hypertensive Rats

5. Six Commercially Available Angiotensin II AT1 Receptor Antibodies are Non-specific

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