Molecular mechanisms underlying fructose‐induced cardiovascular disease: exercise, metabolic pathways and microRNAs

Author:

Kindlovits Raquel12,Bertoldi Julia Maria Cabral Relvas Jacome12,Rocha Helena Naly Miguens12ORCID,Bento‐Bernardes Thais1,Gomes João Lucas Penteado23,Oliveira Edilamar Menezes23,Muniz Ingrid Cristina12,Pereira Juliana Frota12,Fernandes‐Santos Caroline4,Rocha Natália Galito12,Nóbrega Antonio Claudio Lucas da12ORCID,Medeiros Renata Frauches25

Affiliation:

1. Laboratory of Exercise Sciences Department of Physiology and Pharmacology Fluminense Federal University Niterói Rio de Janeiro Brazil

2. National Institute for Science and Technology – INCT Physical (In)activity and Exercise, CNPq – Niterói Rio de Janeiro Brazil

3. Laboratory of Biochemistry and Molecular Biology of Exercise School of Physical Education and Sport University of São Paulo São Paulo Brazil

4. Department of Basic Sciences Fluminense Federal University Nova Friburgo Rio de Janeiro Brazil

5. Department of Nutrition and Dietetics Fluminense Federal University Niterói Rio de Janeiro Brazil

Funder

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Publisher

Wiley

Subject

Physiology,Physiology (medical),Nutrition and Dietetics

Reference49 articles.

1. Respostas metabólicas agudas ao exercício físico moderado em ratos wistar;Afonso M.;Motriz, Rio Claro,2003

2. Antidiabetic effect of hydroalcholic urticadioica leaf extract in male rats with fructose‐induced insulin resistance;Ahangarpour A.;Iranian Journal of Medical Sciences,2012

3. Inactivation of INK4a and ARF induces myocardial proliferation and improves cardiac repair following ischemia-reperfusion

4. Fasting plasma glucose and lipid profiles of diabetic patients improve with aerobic exercise training

5. Cardiac fibroblast‐specific p38α MAP kinase promotes cardiac hypertrophy via a putative paracrine interleukin‐6 signaling mechanism

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