Altered phosphatidylinositol regulation of mutant inwardly rectifying K + Kir7.1 channels associated with inherited retinal degeneration disease
Author:
Affiliation:
1. Centro de Estudios Científicos (CECs) Valdivia Chile
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP280681
Reference45 articles.
1. Defective Potassium Channel Kir2.1 Trafficking Underlies Andersen-Tawil Syndrome
2. Novel dominant K ATP channel mutations in infants with congenital hyperinsulinism: Validation by in vitro expression studies and in vivo carrier phenotyping
3. Kir7.1 inwardly rectifying K+ channel is expressed in ciliary body non pigment epithelial cells and might contribute to intraocular pressure regulation
4. G protein–coupled receptors differentially regulate glycosylation and activity of the inwardly rectifying potassium channel Kir7.1
5. Rapid recycling of ClC-2 chloride channels between plasma membrane and endosomes: Role of a tyrosine endocytosis motif in surface retrieval
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Normal vision and development in mice with low functional expression of Kir7.1 in heterozygosis for a blindness-producing mutation inactivating the channel;American Journal of Physiology-Cell Physiology;2024-04-01
2. The unique structural characteristics of the Kir 7.1 inward rectifier potassium channel: a novel player in energy homeostasis control;American Journal of Physiology-Cell Physiology;2023-03-01
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