G protein–coupled receptors differentially regulate glycosylation and activity of the inwardly rectifying potassium channel Kir7.1
Author:
Funder
HHS NIH National Institute of Diabetes and Digestive and Kidney Diseases
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference39 articles.
1. Inwardly rectifying potassium channels: their structure, function, and physiological roles;Hibino;Physiol. Rev,2010
2. The epithelial inward rectifier channel Kir7.1 displays unusual K+ permeation properties;Döring;J. Neurosci,1998
3. Inwardly rectifying K+ channel Kir7.1 is highly expressed in thyroid follicular cells, intestinal epithelial cells and choroid plexus epithelial cells: implication for a functional coupling with Na+,K+-ATPase;Nakamura;Biochem. J,1999
4. Expression and localization of the inwardly rectifying potassium channel Kir7.1 in native bovine retinal pigment epithelium;Yang;Invest. Opthalmol. Vis. Sci,2003
5. Snowflake vitreoretinal degeneration (SVD) mutation R162W provides new insights into Kir7.1 ion channel structure and function;Pattnaik;PLoS One,2013
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