Cold-induced disruption of Na+channel slow inactivation underlies paralysis in highly thermosensitive paramyotonia
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2008.165787/fullpdf
Reference40 articles.
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2. External pore residue mediates slow inactivation in mu 1 rat skeletal muscle sodium channels;Balser;J Physiol,1996
3. Protein components of the purified sodium channel from rat skeletal muscle sarcolemma;Barchi;J Neurochem,1983
4. A1152D mutation of the Na+ channel causes paramyotonia congenita and emphasizes the role of DIII/S4-S5 linker in fast inactivation;Bouhours;J Physiol,2005
5. Functional characterization and cold sensitivity of T1313A, a new mutation of the skeletal muscle sodium channel causing paramyotonia congenita in humans;Bouhours;J Physiol,2004
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