Compensatory axon sprouting for very slow axonal die-back in a transgenic model of spinal muscular atrophy type III

Author:

Udina Esther12,Putman Charles T.13,Harris Luke R.13ORCID,Tyreman Neil1,Cook Victoria E.13,Gordon Tessa14

Affiliation:

1. Neuroscience and Mental Health Institute, Faculty of Medicine and Dentistry; University of Alberta; Edmonton AB Canada T6G 2S2

2. Institute of Neurosciences and Department of Cell Biology, Physiology and Immunology; Universitat Autònoma de Barcelona, and Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas (CIBERNED); Bellaterra Spain

3. Exercise Biochemistry Laboratory, Faculty of Physical Education and Recreation; University of Alberta; Edmonton AB Canada T6G 2H9

4. Division of Rehabilitation and Physical Medicine of the Faculty of Medicine and Dentistry; University of Alberta; Edmonton AB Canada T6G 2S2

Funder

Spinal Muscular Atrophy Foundation

Natural Sciences and Engineering Research Council of Canada

Publisher

Wiley

Subject

Physiology

Reference76 articles.

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2. Recent advances in amyotrophic lateral sclerosis;Al Chalabi;Curr Opin Neurol,2000

3. Brief electrical stimulation promotes the speed and accuracy of motor axonal regeneration;Al-Majed;J Neurosci,2000

4. Reduced expression of nicotinic AChRs in myotubes from spinal muscular atrophy I patients;Arnold;Lab Invest,2004

5. Histologic and transcriptional assessment of a mild SMA model;Balabanian;Neurol Res,2007

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