Impaired chromaffin cell excitability and exocytosis in autistic Timothy syndrome TS2‐neo mouse rescued by L‐type calcium channel blockers-Reference-Cited by-同舟云学术

Impaired chromaffin cell excitability and exocytosis in autistic Timothy syndrome TS2‐neo mouse rescued by L‐type calcium channel blockers

Published:2019-01-28 Issue:6 Volume:597 Page:1705-1733
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Calorio Chiara¹,Gavello Daniela¹,Guarina Laura¹,Salio Chiara²^ORCID,Sassoè‐Pognetto Marco³^ORCID,Riganti Chiara⁴^ORCID,Bianchi Federico Tommaso⁵,Hofer Nadja T.⁶^ORCID,Tuluc Petronel⁶^ORCID,Obermair Gerald J.⁷,Defilippi Paola⁵,Balzac Fiorella⁵^ORCID,Turco Emilia⁵,Bett Glenna C.⁸,Rasmusson Randall L.⁸,Carbone Emilio¹^ORCID

Affiliation:

1. Department of Drug Science, NIS CentreUniversity of Torino Torino Italy

2. Department of Veterinary SciencesUniversity of Torino Torino Italy

3. Department of Neuroscience Rita Levi MontalciniUniversity of Torino Torino Italy

4. Department of OncologyUniversity of Torino Torino Italy

5. Department of Molecular Biotechnology and Health SciencesUniversity of Torino Torino Italy

6. Department of Pharmacology and Toxicology, Center for Molecular BiosciencesUniversity of Innsbruck Innsbruck Austria

7. Department of Physiology & Medical PhysicsMedical University of Innsbruck Innsbruck Austria

8. Department of Physiology & BiophysicsState University of New York Buffalo NY USA

Funder

Austrian Science Fund

Fondazione Telethon

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP277487

Reference77 articles.

1. Mouse model of Timothy syndrome recapitulates triad of autistic traits

2. Direct and Remote Modulation of L-Channels in Chromaffin Cells: Distinct Actions on α_1C and α_1D Subunits?

3. Autism and induced labor: is calcium a potential mechanistic link?

4. The Timothy syndrome mutation differentially affects voltage- and calcium-dependent inactivation of CaV1.2 L-type calcium channels

5. Nitrendipine block of cardiac calcium channels: high-affinity binding to the inactivated state.

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