JAK/STAT inhibition augments soleus muscle function in a rat model of critical illness myopathy via regulation of complement C3/3R-Reference-Cited by-同舟云学术

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JAK/STAT inhibition augments soleus muscle function in a rat model of critical illness myopathy via regulation of complement C3/3R

Published:2021-05-02 Issue:11 Volume:599 Page:2869-2886
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Addinsall Alex B.¹^ORCID,Cacciani Nicola¹²,Akkad Hazem¹,Salah Heba¹³,Tchkonia Tamara⁴,Kirkland James L.⁴,Larsson Lars¹²^ORCID

Affiliation:

1. Department of Physiology and Pharmacology Karolinska Institutet Stockholm Sweden

2. Department of Clinical Neuroscience Karolinska Institutet Stockholm Sweden

3. Present address: Department of Basic‐Medical Sciences An‐Najah National University Nablus Palestinian Territory

4. Robert and Arlene Kogod Center on Aging Mayo Clinic Rochester MN USA

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP281220

Reference62 articles.

1. Impaired exercise performance is independent of inflammation and cellular stress following genetic reduction or deletion of selenoprotein S

2. Deficiency of selenoprotein S, an endoplasmic reticulum resident oxidoreductase, impairs the contractile function of fast-twitch hindlimb muscles

3. Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis

4. Rapid Determination of Myosin Heavy Chain Expression in Rat, Mouse, and Human Skeletal Muscle Using Multicolor Immunofluorescence Analysis

5. Chaperone co‐inducer BGP‐15 mitigates early contractile dysfunction of the soleus muscle in a rat ICU model

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