OPN‐a induces muscle inflammation by increasing recruitment and activation of pro‐inflammatory macrophages-Reference-Cited by-同舟云学术

OPN‐a induces muscle inflammation by increasing recruitment and activation of pro‐inflammatory macrophages

Published:2016-09-24 Issue:10 Volume:101 Page:1285-1300
ISSN:0958-0670
Container-title:Experimental Physiology
language:en
Short-container-title:Exp Physiol

Author:

Many Gina M.¹²³,Yokosaki Yasuyuki⁴,Uaesoontrachoon Kitipong¹,Nghiem Peter P.¹²⁵,Bello Luca¹,Dadgar Sherry¹,Yin Ying⁶,Damsker Jesse M.¹⁷,Cohen Heather B.⁸,Kornegay Joe N.⁵,Bamman Marcas M.³,Mosser David M.⁸,Nagaraju Kanneboyina¹²,Hoffman Eric P.¹²

Affiliation:

1. Research Center for Genetic Medicine Children's National Medical Center Washington DC USA

2. Department of Integrative Systems Biology George Washington University School of Medicine & Health Sciences Washington DC USA

3. Department of Cell Developmental and Integrative Biology University of Alabama Birmingham Birmingham AL USA

4. Hiroshima University Minamiku Hiroshima Japan

5. Department of Veterinary Integrative Biosciences Texas A&M University College Station TX USA

6. National Institute of Dental and Craniofacial Research National Institutes of Health Bethesda MD USA

7. ReveraGen BioPharma Rockville MD USA

8. Department of Cell Biology and Molecular Genetics University of Maryland College Park MD USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Physiology,Physiology (medical),Nutrition and Dietetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/EP085768

Reference44 articles.

1. Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis

2. Osteopontin as a therapeutic target for cancer

3. Eccentric muscle challenge shows osteopontin polymorphism modulation of muscle damage

4. Genetic modifiers of ambulation in the cooperative international Neuromuscular research group Duchenne natural history study

5. Polymyxin-B inhibition of LPS-induced interleukin-1 secretion by human monocytes is dependent upon the LPS origin

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