FAK Expression, Not Kinase Activity, Is a Key Mediator of Thyroid Tumorigenesis and Protumorigenic Processes-Reference-Cited by-同舟云学术

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FAK Expression, Not Kinase Activity, Is a Key Mediator of Thyroid Tumorigenesis and Protumorigenic Processes

Published:2016-06-03 Issue:9 Volume:14 Page:869-882
ISSN:1541-7786
Container-title:Molecular Cancer Research
language:en
Short-container-title:Mol Cancer Res

Author:

Kessler Brittelle E.,Sharma Vibha,Zhou Qiong,Jing Xia,Pike Laura A.,Kerege Anna A.,Sams Sharon B.,Schweppe Rebecca E.

Funder

NCI

NIHNCINCI

UCDNCINCI

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology,Molecular Biology

Cited by 30 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cancer-associated fibroblast-derived periostin promotes papillary thyroid tumor growth through integrin-FAK-STAT3 signaling;Theranostics;2024

2. Focal adhesion kinase: from biological functions to therapeutic strategies;Experimental Hematology & Oncology;2023-09-25

3. Fibronectin Contributes to a BRAF Inhibitor–driven Invasive Phenotype in Thyroid Cancer through EGR1, Which Can Be Blocked by Inhibition of ERK1/2;Molecular Cancer Research;2023-05-23

4. Recent Developments in Medicinal Chemistry and Therapeutic Potential of Anti-Cancer PROTACs-Based Molecules;Current Medicinal Chemistry;2023-04

5. Focal adhesion kinase inhibition decreases cell viability and induces apoptosis of JAK2 V617F positive cells;Brazilian Journal of Pharmaceutical Sciences;2023

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