Induction of prolonged early G1arrest by CDK4/CDK6 inhibition reprograms lymphoma cells for durable PI3Kδ inhibition through PIK3IP1-Reference-Cited by-同舟云学术

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Induction of prolonged early G1arrest by CDK4/CDK6 inhibition reprograms lymphoma cells for durable PI3Kδ inhibition through PIK3IP1

Published:2013-06-15 Issue:12 Volume:12 Page:1892-1900
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Chiron David,Martin Peter,Di Liberto Maurizio,Huang Xiangao,Ely Scott,Lannutti Brian J.,Leonard John P.,Mason Christopher E.,Chen-Kiang Selina

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

http://www.tandfonline.com/doi/pdf/10.4161/cc.24928

Reference45 articles.

1. The phosphoinositide 3-kinase pathway;Cantley;Science,2002

2. PI3Kδ inhibitors in cancer: rationale and serendipity merge in the clinic;Fruman;Cancer Discov,2011

3. P110delta, a novel phosphoinositide 3-kinase in leukocytes;Vanhaesebroeck;Proc Natl Acad Sci USA,1997

4. PI3 kinase signals BCR-dependent mature B cell survival;Srinivasan;Cell,2009

5. Phosphatidylinositol 3-kinase-δ inhibitor CAL-101 shows promising preclinical activity in chronic lymphocytic leukemia by antagonizing intrinsic and extrinsic cellular survival signals;Herman;Blood,2010

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