The RAX/PACT-PKR stress response pathway promotes p53 sumoylation and activation, leading to G1arrest
Author:
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
http://www.tandfonline.com/doi/pdf/10.4161/cc.11.2.18999
Reference46 articles.
1. RAX, a Cellular Activator for Double-stranded RNA-dependent Protein Kinase during Stress Signaling
2. Interleukin 3 stimulates protein synthesis by regulating double-stranded RNA-dependent protein kinase.
3. Serine 18 Phosphorylation of RAX, the PKR Activator, Is Required for PKR Activation and Consequent Translation Inhibition
4. Replication of Hepatitis C Virus (HCV) RNA in Mouse Embryonic Fibroblasts: Protein Kinase R (PKR)-Dependent and PKR-Independent Mechanisms for Controlling HCV RNA Replication and Mediating Interferon Activities
5. PACT, a Stress-modulated Cellular Activator of Interferon-induced Double-stranded RNA-activated Protein Kinase, PKR
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2. Luteolin protects DYT-PRKRA cells from apoptosis by suppressing PKR activation;Frontiers in Pharmacology;2023-02-15
3. PACT promotes the metastasis of basal-like breast cancer through Rac1 SUMOylation and activation;Oncogene;2022-08-16
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