Werner syndrome protein suppresses the formation of large deletions during the replication of human telomeric sequences
Author:
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
http://www.tandfonline.com/doi/pdf/10.4161/cc.21399
Reference53 articles.
1. Werner and Hutchinson-Gilford progeria syndromes: mechanistic basis of human progeroid diseases;Kudlow;Nat Rev Mol Cell Biol,2007
2. Positional cloning of the Werner’s syndrome gene;Yu;Science,1996
3. The RecQ helicase WRN is required for normal replication fork progression after DNA damage or replication fork arrest;Sidorova;Cell Cycle,2008
4. Defective telomere lagging strand synthesis in cells lacking WRN helicase activity;Crabbe;Science,2004
5. WRN is required for ATM activation and the S-phase checkpoint in response to interstrand cross-link-induced DNA double-strand breaks;Cheng;Mol Biol Cell,2008
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