Profiling beneficial and potential adverse effects of MeCP2 overexpression in a hypomorphic Rett syndrome mouse model-Reference-Cited by-同舟云学术

Profiling beneficial and potential adverse effects of MeCP2 overexpression in a hypomorphic Rett syndrome mouse model

Published:2021-05-28 Issue:1 Volume:21 Page:
ISSN:1601-1848
Container-title:Genes, Brain and Behavior
language:en
Short-container-title:Genes Brain and Behavior

Author:

Vermudez Sheryl Anne D.¹^ORCID,Gogliotti Rocco G.²,Arthur Bright¹,Buch Aditi¹,Morales Clarissa¹,Moxley Yuta¹,Rajpal Hemangi¹,Conn P. Jeffrey¹³⁴,Niswender Colleen M.¹³⁴

Affiliation:

1. Department of Pharmacology and Warren Center for Neuroscience Drug Discovery Vanderbilt University Nashville Tennessee USA

2. Department of Molecular Pharmacology and Neuroscience Loyola University Chicago Chicago Illinois USA

3. Vanderbilt Kennedy Center Vanderbilt University Nashville Tennessee USA

4. Vanderbilt Institute of Chemical Biology Vanderbilt University Nashville Tennessee USA

Funder

National Institute of Mental Health

National Institute of Neurological Disorders and Stroke

National Institutes of Health

Publisher

Wiley

Subject

Behavioral Neuroscience,Neurology,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/gbb.12752

Reference52 articles.

1. Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

2. Reversal of Neurological Defects in a Mouse Model of Rett Syndrome

3. Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice

4. Improved Survival and Reduced Phenotypic Severity Following AAV9/MECP2 Gene Transfer to Neonatal and Juvenile Male Mecp2 Knockout Mice

5. Improved MECP2 Gene Therapy Extends the Survival of MeCP2-Null Mice without Apparent Toxicity after Intracisternal Delivery

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