Inhibin B, AMH, but not INSL3, IGF1 or DHEAS support differentiation between constitutional delay of growth and puberty and hypogonadotropic hypogonadism
Author:
Affiliation:
1. Center of Reproductive Medicine and Andrology, Clinical Andrology; University of Münster; Münster Germany
2. Center of Excellence in Genomic Medicine Research; King Abdulaziz University; Jeddah Saudi Arabia
Publisher
Wiley
Subject
Urology,Endocrinology,Reproductive Medicine,Endocrinology, Diabetes and Metabolism
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/andr.12088/fullpdf
Reference28 articles.
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2. The role of the testicular factor INSL3 in establishing the gonadal position;Adham;Mol Cell Endocrinol,2000
3. Serum inhibin B in healthy pubertal and adolescent boys: relation to age, stage of puberty, and follicle-stimulating hormone, luteinizing hormone, testosterone, and estradiol levels;Andersson;J Clin Endocrinol Metab,1997
4. Inhibin B plus LH vs GnRH agonist test for distinguishing constitutional delay of growth and puberty from isolated hypogonadotropic hypogonadism in boys;Binder;Clin Endocrinol (Oxf),2015
5. Physiological androgen insensitivity of the fetal, neonatal, and early infantile testis is explained by the ontogeny of the androgen receptor expression in Sertoli cells;Chemes;J Clin Endocrinol Metab,2008
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1. Insulin-like peptide 3 (INSL3) as an indicator of leydig cell insufficiency (LCI) in Middle-aged and older men with hypogonadism: reference range and threshold;The Aging Male;2024-04-26
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