Inhibin B, AMH, but not INSL3, IGF1 or DHEAS support differentiation between constitutional delay of growth and puberty and hypogonadotropic hypogonadism-Reference-Cited by-同舟云学术

Inhibin B, AMH, but not INSL3, IGF1 or DHEAS support differentiation between constitutional delay of growth and puberty and hypogonadotropic hypogonadism

Published:2015-08-12 Issue:5 Volume:3 Page:882-887
ISSN:2047-2919
Container-title:Andrology
language:en
Short-container-title:Andrology

Author:

Rohayem J.¹,Nieschlag E.¹²,Kliesch S.¹,Zitzmann M.¹

Affiliation:

1. Center of Reproductive Medicine and Andrology, Clinical Andrology; University of Münster; Münster Germany

2. Center of Excellence in Genomic Medicine Research; King Abdulaziz University; Jeddah Saudi Arabia

Publisher

Wiley

Subject

Urology,Endocrinology,Reproductive Medicine,Endocrinology, Diabetes and Metabolism

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/andr.12088/fullpdf

Reference28 articles.

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3. Serum inhibin B in healthy pubertal and adolescent boys: relation to age, stage of puberty, and follicle-stimulating hormone, luteinizing hormone, testosterone, and estradiol levels;Andersson;J Clin Endocrinol Metab,1997

4. Inhibin B plus LH vs GnRH agonist test for distinguishing constitutional delay of growth and puberty from isolated hypogonadotropic hypogonadism in boys;Binder;Clin Endocrinol (Oxf),2015

5. Physiological androgen insensitivity of the fetal, neonatal, and early infantile testis is explained by the ontogeny of the androgen receptor expression in Sertoli cells;Chemes;J Clin Endocrinol Metab,2008

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