The metabolic impact of β-hydroxybutyrate on neurotransmission: Reduced glycolysis mediates changes in calcium responses and KATPchannel receptor sensitivity-Reference-Cited by-同舟云学术

The metabolic impact of β-hydroxybutyrate on neurotransmission: Reduced glycolysis mediates changes in calcium responses and KATPchannel receptor sensitivity

Published:2015-01-13 Issue:5 Volume:132 Page:520-531
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Lund Trine M.¹,Ploug Kenneth B.²,Iversen Anne¹²,Jensen Anders A.¹,Jansen-Olesen Inger²

Affiliation:

1. Department of Drug Design and Pharmacology; Faculty of Health and Medical Sciences; University of Copenhagen; Copenhagen Denmark

2. Danish Headache Center; Department of Neurology and Research Institute; Glostrup Hospital; University of Copenhagen; Glostrup Denmark

Funder

Aase and Ejnar Danielsens Foundation

Novo Nordisk Foundation

Lundbeck Foundation Center for Neurovascular Signaling (LUCENS)

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12975/fullpdf

Reference36 articles.

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3. Characterization of depolarization-coupled release of glutamate from cultured mouse cerebellar granule cells using DL-threo-beta-benzyloxyaspartate (DL-TBOA) to distinguish between the vesicular and cytoplasmic pools;Bak;Neurochem. Int.,2003

4. Glucose is necessary to maintain neurotransmitter homeostasis during synaptic activity in cultured glutamatergic neurons;Bak;J. Cereb. Blood Flow Metab.,2006

5. Zinc inhibits glutamate release via activation of pre-synaptic K channels and reduces ischaemic damage in rat hippocampus;Bancila;J. Neurochem.,2004

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