The lack of RNA-dependent protein kinase enhances susceptibility of mice to genital herpes simplex virus type 2 infection
Author:
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2567.2006.02403.x/fullpdf
Reference36 articles.
1. Incidence of Herpes Simplex Virus Type 2 Infection in the United States
2. Recurrences after Oral and Genital Herpes Simplex Virus Infection
3. Neutrophils Aid in Protection of the Vaginal Mucosae of Immune Mice against Challenge with Herpes Simplex Virus Type 2
4. Differential roles of B cells and IFN-γ-secreting CD4+ T cells in innate and adaptive immune control of genital herpes simplex virus type 2 infection in mice
5. Interleukin-15 and Natural Killer and NKT Cells Play a Critical Role in Innate Protection against Genital Herpes Simplex Virus Type 2 Infection
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3. The Protein Kinase Double-Stranded RNA-Dependent (PKR) Enhances Protection against Disease Cause by a Non-Viral Pathogen;PLoS Pathogens;2013-08-22
4. Double-Stranded RNA-Activated Protein Kinase Regulates Early Innate Immune Responses During Respiratory Syncytial Virus Infection;Journal of Interferon & Cytokine Research;2010-04
5. HSV Type 2 Infection Increases HIV DNA Detection in Vaginal Tissue of Mice Expressing Human CD4 and CCR5;AIDS Research and Human Retroviruses;2009-11
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