Novel FLT3 point mutations within exon 14 found in patients with acute myeloid leukaemia
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2004.04808.x/fullpdf
Reference12 articles.
1. Identification of novel FLT-3 Asp835 mutations in adult acute myeloid leukaemia;Abu-Duhier;British Journal of Haematology,2001
2. A repressor sequence in the juxtamembrane domain of Flt-1 (VEGFR-1) constitutively inhibits vascular endothelial growth factor-dependent phosphatidylinositol 3′-kinase activation and endothelial cell migration;Gille;EMBO Journal,2000
3. Prognostic implication of FLT3 and N-RAS gene mutations in acute myeloid leukemia;Kiyoi;Blood,1999
4. The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the United Kingdom Medical Research Council AML 10 and 12 trials;Kottaridis;Blood,2001
5. Prevalence and prognostic significance of FLT3 internal tandem duplication in pediatric acute myeloid leukemia;Meshinchi;Blood,2001
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2. Clinical and molecular features of FLT3 juxtamembrane domain missense mutations in acute myeloid leukaemia;Journal of Cellular and Molecular Medicine;2022-11-28
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