Kisspeptin/GPR54 system as potential target for endocrine disruption of reproductive development and function
Author:
Publisher
Wiley
Subject
Urology,Reproductive Medicine,Endocrinology, Diabetes and Metabolism
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2605.2009.01012.x/fullpdf
Reference53 articles.
1. The Creb1 coactivator Crtc1 is required for energy balance and fertility;Altarejos;Nature Medicine,2008
2. Disrupted female reproductive physiology following neonatal exposure to phytoestrogens or estrogen specific ligands is associated with decreased GnRH activation and kisspeptin fiber density in the hypothalamus;Bateman;Neurotoxicology,2008
3. Exposure to a complex cocktail of environmental endocrine disrupting compounds disturbs the Kiss1/GPR54 system in ovine hypothalamus and pituitary gland;Bellingham;Environmental Health Perspectives,2009
4. KiSS-1/kisspeptins and the metabolic control of reproduction: physiologic roles and putative physiopathological implications;Castellano;Peptides,2009
5. Postnatal development of kisspeptin neurons in mouse hypothalamus; sexual dimorphism and projections to gonadotropin-releasing hormone neurons;Clarkson;Endocrinology,2006
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