Celebrities in the heart, strangers in the pancreatic beta cell: Voltage‐gated potassium channels K v 7.1 and K v 11.1 bridge long QT syndrome with hyperinsulinaemia as well as type 2 diabetes
Author:
Affiliation:
1. Department of Biomedical Sciences Faculty of Health and Medical Sciences University of Copenhagen Copenhagen Denmark
Funder
Novo Nordisk Fonden
Hjerteforeningen
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/apha.13781
Reference171 articles.
1. Electrophysiology of the pancreas.
2. Coassembly of KVLQT1 and minK (IsK) proteins to form cardiac IKS potassium channel
3. Voltage-dependent K + channels in pancreatic beta cells: Role, regulation and potential as therapeutic targets
4. KCNQ1 Long QT Syndrome Patients Have Hyperinsulinemia and Symptomatic Hypoglycemia
5. Patients With Long-QT Syndrome Caused by Impaired hERG -Encoded K v 11.1 Potassium Channel Have Exaggerated Endocrine Pancreatic and Incretin Function Associated With Reactive Hypoglycemia
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2. Potassium voltage-gated channel subfamily H member 2 (KCNH2) is a promising target for incretin secretagogue therapies;Signal Transduction and Targeted Therapy;2024-08-12
3. Hormones—Modulators of metabolism;Acta Physiologica;2023-06-08
4. Pancreatic Beta-cell Dysfunction in Type 2 Diabetes;European Journal of Inflammation;2023-01-30
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