PI(3,4)P2 plays critical roles in the regulation of focal adhesion dynamics of MDA-MB-231 breast cancer cells-Reference-Cited by-同舟云学术

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PI(3,4)P2 plays critical roles in the regulation of focal adhesion dynamics of MDA-MB-231 breast cancer cells

Published:2017-05 Issue:5 Volume:108 Page:941-951
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Sci

Author:

Fukumoto Miki¹,Ijuin Takeshi²^ORCID,Takenawa Tadaomi³

Affiliation:

1. The Integrated Center for Mass Spectrometry; Kobe University Graduate School of Medicine; Kobe Japan

2. Division of Biochemistry; Kobe University Graduate School of Medicine; Kobe Japan

3. Division of Molecular and Cell Biology; Kobe University Graduate School of Medicine; Kobe Japan

Funder

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/cas.13215/fullpdf

Reference53 articles.

1. Steering cell migration: lamellipodium dynamics and the regulation of directional persistence;Krause;Nat Rev Mol Cell Biol,2014

2. Profilin1 regulates PI(3,4)P2 and lamellipodin accumulation at the leading edge thus influencing motility of MDA-MB-231 cells;Bae;Proc Natl Acad Sci USA,2010

3. Lamellipodin and the Scar/WAVE complex cooperate to promote cell migration in vivo;Law;J Cell Biol,2013

4. PI3K pathway activation in breast cancer is associated with the basal-like phenotype and cancer-specific mortality;Lopez-Knowles;Int J Cancer,2010

5. The Cancer Genome Atlas: clinical applications for breast cancer;Ma;Oncology (Williston Park),2013

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