Manganese neurotoxicity: behavioral disorders associated with dysfunctions in the basal ganglia and neurochemical transmission

Author:

Bouabid Safa123,Tinakoua Anass123,Lakhdar-Ghazal Nouria3,Benazzouz Abdelhamid12

Affiliation:

1. University de Bordeaux; Institut des Maladies Neurodégénératives; UMR 5293 Bordeaux France

2. CNRS; Institut des Maladies Neurodégénératives; UMR 5293 Bordeaux France

3. Université Mohammed V; Faculté des Sciences; Equipe Rythmes Biologiques; Neurosciences et Environnement; Rabat Morocco

Funder

Université de Bordeaux

Université Mohammed 5

Centre National de la Recherche Scientifique

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Reference192 articles.

1. The chromium, manganese, and cobalt complexes of transferrin;Aisen;J. Biol. Chem.,1969

2. Functional architecture of basal ganglia circuits: neural substrates of parallel processing;Alexander;Trends Neurosci.,1990

3. Pharmacokinetic data needs to support risk assessments for inhaled and ingested manganese;Andersen;Neurotoxicology,1999

4. Inhibition of DAT function attenuates manganese accumulation in the globus pallidus;Anderson;Environ. Toxicol. Pharmacol.,2007

5. Manganese exposure alters extracellular GABA, GABA receptor and transporter protein and mRNA levels in the developing rat brain;Anderson;Neurotoxicology,2008

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