The effect of inflammation on Fos expression in the ferret trigeminal nucleus
Author:
Publisher
Wiley
Subject
General Dentistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0722.2007.00411.x/fullpdf
Reference66 articles.
1. Acute and Chronic Craniofacial Pain: Brainstem Mechanisms of Nociceptive Transmission and Neuroplasticity, and Their Clinical Correlates
2. Dental Injury Models: Experimental Tools for Understanding Neuroinflammatory Interactions and Polymodal Nociceptor Functions
3. Effects of injury and inflammation on pulpal and periapical nerves
4. Neural Changes in Periapical Lesions after Systemic Steroids in the Ferret
5. Responses of nerve fibers to pulpal inflammation and periapical lesions in rat molars demonstrated by calcitonin gene-related peptide immunocytochemistry
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1. Dental Pain Mechanisms;The Senses: A Comprehensive Reference;2020
2. Chronic tooth pulp inflammation induces persistent expression of phosphorylated ERK (pERK) and phosphorylated p38 (pp38) in trigeminal subnucleus caudalis;Neuroscience;2014-06
3. Evidence for anti-inflammatory and putative analgesic effects of a monoclonal antibody to calcitonin gene-related peptide;Neuroscience;2013-01
4. Expression changes of K+–Cl− co-transporter 2 and Na+–K+–Cl− co-transporter1 in mouse trigeminal subnucleus caudalis following pulpal inflammation;Brain Research Bulletin;2010-04
5. The effects of ibuprofen and the neurokinin-1 receptor antagonist GR205171A on Fos expression in the ferret trigeminal nucleus following tooth pulp stimulation;European Journal of Pain;2008-04
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